[[youtube](https://www.youtube.com/watch?v=_q44phEpFjA)] Click here if you need help quitting psychiatric medications: https://membership.taperclinic.com/signup Why Jordan Peterson’s Benzo Story Could Happen to You Using Dr. Peterson’s own public history, I explain how long-term use of benzodiazepines (such as Xanax, Klonopin, and Valium) can, in some patients, lead to a severe neurological condition now known as Benzodiazepine-Induced Neurological Dysfunction (BIND). We walk through: • What benzodiazepines actually do to the brain • Why they are not meant for long-term use • How dependence quietly forms even at “low” doses • The symptoms of BIND and protracted withdrawal • How these reactions are often misdiagnosed as worsening mental illness • Why rapid tapers and detoxes can permanently injure the nervous system • The tragic medical decisions that followed • Why recovery often takes 18–24 months • And why this can happen to everyday patients, not just public figures I also discuss the controversial and dangerous use of propofol (the same anesthetic involved in the death of Michael Jackson) in so-called “rapid detox” clinics, and why slow, patient-led tapering is the only evidence-based approach for a sensitized nervous system. There is currently no test to predict who will develop BIND. That’s why this matters. If you or someone you love has been prescribed benzodiazepines for anxiety, sleep, or stress, this video could change how you think about these drugs forever. If this helped you, consider subscribing. My goal is simple: prevent avoidable medication injuries by telling the truth most patients are never told. Get our free tapering course: https://membership.taperclinic.com/free-taper-guide Join +6000 people learning how to taper on our newsletter: https://membership.taperclinic.com/newsletter CONNECT WITH ME: Instagram: / taperclinic TikTok: / taperclinic X/Twitter: https://x.com/DrJosefWD Website: https://taperclinic.com/ --- The video explains how long‑term benzodiazepine use, and especially rapid withdrawal, caused a severe neurological injury (BIND) in Jordan Peterson, and uses his story to warn that similar harm can occur in ordinary patients and to argue for slow, patient‑led tapering instead of rapid detox approaches.​ ## Detailed outline of the video ## Introduction and thesis - Presenter (Dr. Josef) introduces himself as a psychiatrist and adverse‑effects expert, then states that Jordan Peterson is currently very ill and that part of this may stem from a severe adverse reaction to benzodiazepines in 2020.​ - He outlines the plan: explain what benzodiazepines do, why long‑term use is risky, what BIND is, how it appeared in Peterson’s case, and what viewers should know to reduce their own risk.​ ## Benzodiazepines and their general risks - Defines benzodiazepines as sedative medications that slow the brain, giving examples like Xanax, Klonopin, and Valium, usually prescribed for anxiety, sleep problems, and depression.​ - Emphasizes that they cause physiological dependence, are not meant for long‑term use, and are among the most commonly prescribed psychiatric medications, especially in older adults.​ ## How Peterson started benzodiazepines - Plays a Joe Rogan clip where Peterson describes longstanding depression, high stress around 2016 (job threats, regulatory investigations, a complaint from a client), seasonal affective disorder, and severe insomnia.​ - Peterson explains that his doctor prescribed a sleeping pill plus an anti‑anxiety drug; taking a small dose of clonazepam (0.25 mg twice daily) allowed him to sleep and function, so he continued it, assuming it was safe.​ ## Early toxicity and the concept of BIND - Narrator stresses that Peterson’s decision to stay on the medication is understandable given his history and prior successful antidepressant use.​ - Introduces Benzodiazepine‑Induced Neurological Dysfunction (BIND) as a form of neurotoxicity from long‑term benzo use in a susceptible minority, likening it to chemotherapy‑like damage where neurons are harmed and symptoms worsen over time.​ ## Medical literature on BIND and symptom profile - Shows a recent academic article (Ritvo et al., 2023) and notes that BIND has only recently gained wider recognition and was only added to drug labels in 2021.​ - Reviews survey data of about 1,200 patients, highlighting long‑lasting symptoms such as low energy for years, akathisia (need to move), balance problems, cardiovascular irregularities, stabbing or burning pains, neuropathic symptoms in hands, severe sleep disturbance, cognitive issues, and sensory sensitivities.​ ## Peterson’s worsening symptoms on benzos - Plays a clip where Peterson recalls left‑sided weakness, emotional detachment, reduced capacity for joy, and later severe anxiety that he only later associated with benzodiazepine use.​ - When his wife is diagnosed with a presumed terminal cancer, his anxiety rises; increasing his benzo dose paradoxically worsens anxiety, which the narrator identifies as a sign of drug toxicity rather than under‑treatment.​ ## Smoldering toxicity and misinterpretation as primary illness - Narrator explains that long‑term toxicity can leave the nervous system unstable; attempts to increase the dose or taper can both worsen symptoms.​ - Notes that many clinicians mislabel such cases as “treatment‑resistant depression” rather than recognizing BIND or protracted withdrawal, which only recently appeared in official labeling.​ ## Rapid taper for ketamine and acute withdrawal - Describes how clinicians wanted to try ketamine for presumed refractory depression but had to remove benzodiazepines first because benzos may blunt ketamine’s effect.​ - Reports that Peterson was taken off benzos over about a week to enable ketamine, leading to a dramatic worsening consistent with severe acute withdrawal layered on top of prior toxicity.​ ## Drug labels and protracted withdrawal warnings - Shows the updated Xanax XR label, noting that protracted withdrawal or BIND can last more than 12 months and include anxiety, cognitive impairment, depression, formication, motor symptoms, muscle weakness, tremor, paresthesia, tinnitus, and more.​ - Argues that despite such warnings, many doctors still fail to recognize protracted withdrawal and instead pursue aggressive interventions.​ ## Akathisia and extreme subjective suffering - Plays a clip where Peterson describes stopping and tapering as unbearable, with anxiety higher than anything he’d experienced and severe akathisia—constant internal electric‑like prodding, inability to sit or lie still, and relentless agitation.​ - Narrator elaborates that he has seen patients with continuous electric shocks, jolting, cognitive impairment, and catastrophic thinking for months to a year, noting the high risk of suicidality in this state.​ ## Failed rehab and benzo substitution - Peterson recounts attending a U.S. clinic promising rapid detox, which instead substituted one benzo for another (e.g., clonazepam to diazepam/Valium following an Ashton‑style protocol), leaving him worse and adding more sedatives to suppress akathisia.​ - Narrator differentiates between ordinary dependence and Peterson’s case, emphasizing that once neurons are damaged and the nervous system is sensitized, swapping or stacking sedatives often exacerbates problems, not resolves them.​ ## Escalating attempts at treatment and international travel - The video describes Peterson’s continued deterioration, further hospitalization in Toronto (described as “worse than useless”), and his family’s eventual decision to seek care overseas.​ - Explains that desperation and panic drive families in such situations to try increasingly extreme options, including expensive rehab and overseas treatment.​ ## Propofol‑based “rapid detox” in Russia/Serbia - Peterson and Michaela describe traveling to a Russian/Serbian clinic that used propofol (plus another sedative) to keep him unconscious for about nine days while removing the benzodiazepine.​ - Narrator strongly criticizes this approach, noting that propofol is the anesthetic involved in Michael Jackson’s death and arguing that such deep‑sedation rapid detox is dangerous and not recommended for BIND‑type injuries.​ ## Two‑year recovery trajectory and residual deficits - Plays a Joe Rogan clip where Peterson says recovery from benzos took about two years and that he still is not fully recovered, with persistent numbness in hands and feet and memory of excruciating pain.​ - Narrator frames this as consistent with BIND being akin to traumatic brain injury: the drug is gone, but neurological damage leads to prolonged waves of pain and dysfunction.​ ## Waves, windows, and long‑term sensitivity - Explains the “waves and windows” pattern after the worst acute phase: months of severe symptoms gradually give way to alternating periods of relative wellness and relapse over 18–24 months.​ - Notes that even after overall improvement, patients often remain neurologically sensitive, with later triggers (medications, infections, mold) capable of re‑provoking neurological dysfunction.​ ## Later setback: mold exposure, infection, and ICU stay - Plays Michaela’s update that Peterson recently had pneumonia and sepsis, plus neurological issues thought to be related to chronic inflammatory response syndrome (CIRS) from long‑term mold exposure, and that he was diagnosed with critical illness polyneuropathy or myopathy.​ - Narrator speculates, based on his clinical experience, that prior benzo‑induced sensitization may make patients more reactive to triggers like mold or antibiotics, contributing to such setbacks.​ ## Impact on life, relationships, and work - Cites survey data in which more than half of BIND sufferers report serious impact on marriage/relationships, frequent suicidal thoughts, and major work disruption or job loss, along with increased medical costs.​ - Connects this to Peterson’s hiatus from work and emphasizes that BIND is functionally comparable to a traumatic brain injury in its life‑disrupting effects.​ ## Prognosis and family support role - Emphasizes that many patients only survive such injuries because family and friends “carry them across the finish line,” as many feel they cannot endure the suffering.​ - Notes that, according to Michaela’s later comments, Peterson has shown improvement from his recent setback, and the narrator expresses guarded optimism about his longer‑term prognosis based on patterns seen in other patients.​ ## Final warnings and practical advice - Stresses that while many people use benzodiazepines for years without catastrophic problems, a vulnerable minority can develop BIND, and there is currently no test to predict susceptibility.​ - Concludes by advising viewers not to use benzodiazepines longer than 2–4 weeks when possible, to prioritize non‑drug strategies for anxiety and insomnia, and to use slow, patient‑led tapering rather than rapid detox if discontinuation is needed.​ ## Drugs mentioned, primary uses, and side‑effects | Drug / class | Primary uses in the video | Key side‑effects or risks described | | -------------------------------------------- | ---------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------- | ------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------------ | | **Benzodiazepines** (class overall) | Used for anxiety, sleep problems, stress, and sometimes depression.​ | Physiological dependence after weeks of use; withdrawal and protracted withdrawal (BIND); neurotoxicity in a minority with progressive neurological injury; severe anxiety and insomnia on rapid taper; akathisia; balance problems; cardiovascular issues; neuropathic pain; numbness; cognitive impairment; sensory sensitivity; long recovery (18–24 months); high suicidality risk.​ | | **Clonazepam** (Klonopin, “Clonopin”) | Anti‑anxiety medication and sleep aid for Peterson at 0.25 mg twice daily, long‑acting benzo.​ | Over time associated (in his case) with weakness, emotional detachment, reduced joy, severe anxiety; paradoxical worsening of anxiety at higher doses suggesting toxicity; intense withdrawal and akathisia when tapered or stopped; contribution to BIND.​ | | **Alprazolam** (Xanax, Xanax XR) | Example of a commonly prescribed benzodiazepine; Xanax XR label used to illustrate protracted withdrawal warnings.​ | Label now warns that protracted withdrawal symptoms can last more than 12 months, including anxiety, cognitive impairment, depression, formication, motor symptoms, muscle weakness, tremor, paresthesia, and tinnitus.​ | | **Diazepam** (Valium) | Another common benzodiazepine; used in Ashton‑style protocol and in some clinics to substitute for shorter‑acting benzos in tapering.​ | As a long‑acting benzo, can be somewhat easier to taper for some, but in BIND cases substitution does not solve underlying neurological injury; continued toxicity and withdrawal‑like symptoms may persist.​ | | **Imovane** (zopiclone; non‑benzo hypnotic) | Prescribed to Peterson as a sleeping medication alongside the anti‑anxiety benzo early in his treatment.​ | Not detailed extensively; implied sedative–hypnotic with potential for dependence, but main focus is that once clonazepam worked, he no longer needed Imovane.​ | | **Barbiturates** (class, historical) | Older sedative–anxiolytics that benzos largely replaced; discussed in contrast to benzodiazepines.​ | More likely to cause respiratory depression and fatal overdose than benzos; however, benzos have far worse withdrawal and protracted neurological injury risk in susceptible individuals.​ | | **Ketamine** | Proposed treatment for “treatment‑resistant depression” in Peterson once his doctors thought benzos were not working.​ | Cannot be effectively used while on benzos because benzos may block its effect; attempts to stop benzos quickly to allow ketamine led to severe withdrawal and worsening of Peterson’s condition.​ | | **Propofol** | Powerful anesthetic used in a Russian/Serbian clinic to keep Peterson unconscious for about nine days during a “rapid detox.”​ | Described as controversial and dangerous; same anesthetic involved in Michael Jackson’s death; using it for rapid benzo detox is portrayed as risky and not evidence‑based for BIND.​ | | “Two more sedative‑like drugs” (unspecified) | Added in a rehab setting in an attempt to dampen akathisia after benzo substitution.​ | In a sensitized nervous system with damaged neurons, new medications frequently cause side‑effects and may worsen symptoms rather than relieve them.​ | | **Antibiotics** (class, unspecified) | Mentioned as possible contributors to later setbacks during pneumonia treatment in patients with benzo‑sensitized nervous systems.​ | Suggested that antibiotics can sometimes exacerbate neurological symptoms in highly sensitized patients, though this is presented as clinical speculation rather than proven mechanism.​ | Is there a particular drug from the video you’d like a deeper dive on (e.g., clonazepam vs Xanax pharmacology, or propofol risks)?